Growth & GHPeptideEvidence C4 cited

GHRP-2

Growth Hormone Releasing Peptide-2

aka Growth Hormone Releasing Peptide 2 · GHRP2 · Pralmorelin · KP-102

A second-generation GHS-R agonist that research associates with significant pulsatile growth hormone release.

Molecular wt
818.0g/mol
Formula
C45H55N9O6
CAS
158861-67-7
Half-life
2 h
clinical
Tmax
30 min
Route
Subcutaneous injection

Sequence

Read this before quoting the sequence
Synthetic hexapeptide amide, INN pralmorelin (developer code KP-102). Contains three D-amino acids and the non-proteinogenic residue 3-(2-naphthyl)-D-alanine (D-2-Nal), so a one-letter string would be wrong. PubChem systematic name for CID 6918245: 'L-Lysinamide, D-alanyl-3-(2-naphthalenyl)-D-alanyl-L-alanyl-L-tryptophyl-D-phenylalanyl-'.
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Mechanism of action

GHRP-2 works by binding to the growth hormone secretagogue receptor (GHS-R) on pituitary cells, stimulating the release of growth hormone (GH). It mimics ghrelin, a natural hormone that regulates appetite and energy balance, thereby increasing GH levels. This peptide also influences the hypothalamus to enhance GH secretion.

What the evidence actually shows

Grade C

There are limited human trials on GHRP-2. Some studies suggest it increases GH levels and may improve body composition, but comprehensive clinical trials are lacking. More research is needed to confirm its efficacy and safety in humans.

Regulatory status

Status
Not approved in the US or EU. No entry in Drugs@FDA or the EMA medicines register. In Japan the GHRP-2 test is an available and recommended growth-hormone stimulation test for adult GH deficiency (Fukuda 2014: 'in Japan, the growth hormone releasing peptide-2 (GHRP-2) test is available and is recommended as a convenient and safe GH stimulating test'; the diagnostic cut-off for severe adult GHD is a peak GH of 9 micrograms/L).
Approved as
Diagnostic GH-stimulation agent in Japan (pralmorelin). Therapeutic approval: none anywhere.
Clinical stage
Marketed as a diagnostic agent in Japan; no active therapeutic development programme identified.
WADA
Prohibited at all times. WADA 2026 Prohibited List S2.2.4 names 'GH-releasing peptides (GHRPs) [e.g. alexamorelin, examorelin (hexarelin), GHRP-1, GHRP-2 (pralmorelin), GHRP-3, GHRP-4, GHRP-5 and GHRP-6]'.
UK
research_compound

In the UK, GHRP-2 is classified as a research compound and is not approved for medical use by the MHRA.

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Pharmacokinetics

Half-life
2 h
Tmax
30 min
Absorption
subcutaneous
Elimination
renal
Washout
2 days
Low-confidence pharmacokinetics
These figures are recorded as clinical rather than measured in a published PK study. Treat them as an order of magnitude. Anything downstream of them — accumulation, steady state, washout — inherits that uncertainty.

Dosing — what backs each figure

GHRP-2 (pralmorelin) has genuine published human data, but almost all of it is single-dose GH-stimulation testing: 100 microg intravenously as a validated diagnostic provocation test for adult GH deficiency, or 1 microg/kg intravenously / 1 microg/kg/h subcutaneously in physiology studies. No repeated therapeutic dosing regimen has been established, and chronic administration studies explicitly warn that GH responses desensitise depending on dose and frequency.

ClinicalApproved labelling or a published human trial
Commonly circulatedNot established by any study
Dose
Research literature cites ~100 to 300 mcg per dose
Frequency
2-3x daily

Why this isn't evidence
The real human literature is a single 100 microg INTRAVENOUS bolus used as a one-off GH-deficiency diagnostic test, or weight-based 1 microg/kg IV / 1 microg/kg/h SC infusion - nobody has published a 100-300 microg subcutaneous dose given two or three times a day, and the '4-6 weeks on, then a break' desensitisation cycle in the notes is community convention.

This figure is shown because it is what circulates and what people search for — not because it is supported. Cycle lengths in particular tend to be convention copied between compounds rather than anything derived from a compound's own pharmacokinetics.

Handling and storage

No compound-specific stability data exists
GHRP-2 is used as a diagnostic agent in Japan, but the Japanese product labelling could not be retrieved, and there is no FDA or EMA label. No compound-specific storage specification could be verified from an authoritative source. Only generic lyophilised-peptide handling guidance would otherwise apply. General peptide handling still applies — reconstitute gently, keep it cold, keep it dark — but any specific figure you see quoted for this compound elsewhere is someone's assumption, not a measurement.

Safety

Commonly reported
  • ·Increased appetite
  • ·Water retention
  • ·Joint pain
Rarely reported
  • ·Carpal tunnel syndrome
  • ·Gynecomastia
Contraindications
  • ·Pregnancy
  • ·"Active cancer"
Drug interactions
  • ·Corticosteroids, which may blunt GH release

Increased appetite, water retention, transient prolactin/cortisol rise

Reported combinations

Reported as synergistic
  • ·CJC-1295, which may enhance GH release
Reported as antagonistic
  • ·Somatostatin analogs, which inhibit GH release

Combination claims in this dataset are largely unsourced and should be treated as folk knowledge until a citation appears beside them.

References (4)

Data provenance

Chemistry and citations on this page were checked against primary sources on 2026-08-14. Values that could not be verified were left blank rather than filled with a plausible guess.

Could not verify
storage, reconstitution

Research use only
This profile is educational reference material. It is not medical advice, not a prescription, and not an endorsement of self-administration. Compounds discussed here are research chemicals and most are not approved for human therapeutic use.