Metabolic & GLP-1Evidence A2 cited

Setmelanotide

aka Imcivree · RM-493 · MC4R agonist

Setmelanotide is a synthetic cyclic peptide that acts as a selective agonist of the melanocortin 4 receptor (MC4R). It was FDA-approved in 2020 under the brand name Imcivree for the treatment of chronic weight management in patients with obesity due to proopiomelanocortin (POMC), proprotein convertase subtilisin/kexin type 1 (PCSK1), or leptin receptor (LEPR) deficiency confirmed by genetic testing. By restoring MC4R pathway signalling that is disrupted in these rare genetic obesity conditions, setmelanotide reduces hyperphagia and promotes clinically significant weight loss. It represents a precision medicine approach to obesity, targeting the specific molecular defect rather than broadly suppressing appetite. Clinical trials demonstrated sustained weight reduction in the indicated populations with an acceptable safety profile.

Molecular wt
1117.3g/mol
Formula
C49H68N18O9S2
CAS
920014-72-8
Half-life
11 h
established
Tmax
8 h
Route
Subcutaneous injection

Sequence

Read this before quoting the sequence
Cyclic 8-residue analogue of alpha-melanocyte stimulating hormone, closed by a disulfide bond between the cysteines at positions 2 and 8. The IMCIVREE label gives the chemical name as 'Acetyl-L-arginyl-L-cysteinyl-D-alanyl-L-histidinyl-D-phenylalanyl-L-arginyl-L-tryptophanyl-L-cysteinamide cyclic (2->8)-disulfide acetate'. A one-letter string is not given because the peptide contains two D-amino acids and is cyclic.
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Mechanism of action

Setmelanotide binds to and activates the melanocortin 4 receptor (MC4R) in the hypothalamus, restoring impaired MC4R signalling that regulates hunger and energy expenditure. In patients with POMC, PCSK1, or LEPR deficiency, normal production of alpha-MSH or its upstream signalling is disrupted, leading to severe hyperphagia and early-onset obesity. Setmelanotide bypasses these upstream defects by directly stimulating MC4R, thereby reducing hunger drive and increasing satiety signalling. This targeted mechanism normalises energy balance in genetically susceptible individuals.

What the evidence actually shows

Grade A

Setmelanotide has been evaluated in multiple Phase 3 clinical trials demonstrating significant reductions in hunger and body weight in patients with POMC, PCSK1, and LEPR deficiency. The pivotal trials showed that approximately 80% of POMC-deficient patients and 45% of LEPR-deficient patients achieved at least 10% weight loss. FDA approval was granted in 2020 based on robust clinical evidence. Long-term extension studies continue to support sustained efficacy and acceptable safety profiles.

Regulatory status

Status
Approved prescription medicine (US).
Approved as
IMCIVREE (setmelanotide) injection, Rhythm Pharmaceuticals. Labelled to reduce excess body weight in acquired hypothalamic obesity (age 4 and older), Bardet-Biedl syndrome (age 2 and older) and POMC, PCSK1 or LEPR deficiency (age 2 and older). Label doses: starting 0.5 mg daily in acquired hypothalamic obesity and 2 mg daily from age 12 in Bardet-Biedl syndrome, with a 3 mg daily maintenance dose.
Clinical stage
Marketed / approved; Phase 4 and further Phase 3 studies ongoing.
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Pharmacokinetics

Half-life
11 h
Tmax
8 h
Absorption
subcutaneous
Elimination
hepatic
Washout
7 days
Titration
Start 2mg daily may increase to 3mg
Low-confidence pharmacokinetics
These figures are recorded as established rather than measured in a published PK study. Treat them as an order of magnitude. Anything downstream of them — accumulation, steady state, washout — inherits that uncertainty.

Dosing — what backs each figure

Setmelanotide is FDA-approved as IMCIVREE with a fully specified once-daily subcutaneous regimen: a 2-week starting dose that varies by age and indication (0.5 mg, 1 mg or 2 mg) followed by a 3 mg daily maintenance dose, with weight-based dosing in the youngest patients and reductions in severe renal impairment. It is indicated only for acquired hypothalamic obesity, Bardet-Biedl syndrome and genetically confirmed POMC/PCSK1/LEPR deficiency, and the label explicitly excludes general obesity.

ClinicalApproved labelling or a published human trial
  • Acquired hypothalamic obesity, age 4 and older: 0.5 mg subcutaneously once daily for 2 weeks, then titrate to a 3 mg once-daily maintenance dose (ages 4-6 dosed by body weight)

    adults and pediatric patients aged 4 years and older with acquired hypothalamic obesity · subcutaneous

    FDA label IMCIVREE (setmelanotide) injection, NDA 213793

  • Bardet-Biedl syndrome or POMC/PCSK1/LEPR deficiency: age 12+, 2 mg once daily for 2 weeks then 3 mg maintenance; ages 6-12, 1 mg once daily for 2 weeks then 3 mg maintenance; ages 2-6, 0.5 mg once daily for 2 weeks then a weight-determined maintenance dose. Reduced dosing in severe renal impairment; not recommended in end-stage renal disease

    adults and pediatric patients aged 2 years and older with Bardet-Biedl syndrome, or genetically confirmed POMC, PCSK1 or LEPR deficiency · subcutaneous

    FDA label IMCIVREE, section 2

Handling and storage

Lyophilized
Not applicable — IMCIVREE is supplied as a solution in multiple-dose vials, not lyophilised. Unopened vials are stored refrigerated at 2-8 degC (36-46 degF); they may be kept at room temperature for up to 30 days before the expiration date.
Reconstituted
After the vial is punctured (opened), discard after 30 days.

No light-protection requirement is stated in the IMCIVREE prescribing information, and no freeze-thaw tolerance is claimed.

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Safety

References (2)

Data provenance

Chemistry and citations on this page were checked against primary sources on 2026-08-14. Values that could not be verified were left blank rather than filled with a plausible guess.

Could not verify
regulatory.wada, storage.lightSensitive

Research use only
This profile is educational reference material. It is not medical advice, not a prescription, and not an endorsement of self-administration. Compounds discussed here are research chemicals and most are not approved for human therapeutic use.